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Similarly, another study demonstrated that CU inhibited the progression of PTZ ignition in mice in a dose-dependent manner by improving the levels of MDA and glutathione ( 3 -induced seizures, likely due to curcumin’s significant activation of Na + , K + -ATPase activity and its ability to inhibit lipid and cellular protein damage (Jyoti et al., 2009)

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4.1.3 NF-κB The NF-κB pathway can be activated by the binding of Toll-like receptors to ligands such as TNF and IL-1, serving as a master regulator of inflammation (Yu et al., 2020)